Studies in cardiac pathology (1911) (14757850846)

Identifier: studiesincardiac00norr (find matches)

Title: Studies in cardiac pathology

Year: 1911 (1910s)

Authors: Norris, George William, 1875-1965

Subjects: Cardiovascular System

Publisher: Philadelphia : Saunders

Contributing Library: Columbia University Libraries

Digitizing Sponsor: Open Knowledge Commons

Text Appearing Before Image:

arch ofthe aorta was the seat of large aneurismal formations. The valvesshowed the same rubbery thickening when extensively involved,and occasionally there were crescentic lines of whitish-yellowthickening on the endocardium of the ventricle beneath thevalves. The diagnoses in these cases were corroborated by mi-croscopic examinations, and extension downward into the aortic For further information see Mracek, Arch. f. Dermat. u. Syphilis, xxv, 1893, Ergan-zungsheft, p. 279; Landois, ibid., 1908, p. 221; and Stockmann, Ueber Gummiknoten inHerzfleische bei Erwachsenen, Wiesbaden, 1904. -Warthin: Trans. Assoc. Am. Phys., 1910. Bruhns: BerUn. klin. Woch., xUi, No, 8. ■I Wiesner: Centralbl. f. Path. u. path. Anat., 1905, p. 822. ^ Klotz: Jour. Path, and Bact., 1907, p. 11. Renter; Mijnch. med. Woch., 1906, p. 778; Ztsch. f. Hyg. u. Infektionskh. 1906, p. 49.Benda: BerUn. khn. Woch., 1906, p. 989. Schmorl: Munch, med. Woch., 1907, p. 188.Wright: Boston Med. and Surg. Jour., 1909, p. 539.

Text Appearing After Image:

Fig. 72.—Syphilitic Aortitis.In thi.s specimen the syphilitic prooes.s has extended downward along the aortic wall andinvolved the coronary orifices and the aortic leaflets. The smooth, more or less sharply cir-cumscribed rubbery appearance of the lesions is shown. Also the absence of calcification.(Compare with Fiss. 41, 66, and 71. See also under Cardiac Syphilis, p. 200, and Dis-easf» of the .Vonif Orififo, p. 50.) 204 STUDIES IN CARDIAC PATHOLOGY leaflets suggested that the agent which produced the chronicinflammatory and proliferative changes in the wall of the aortahad some part in the disease of the aortic valves. Of these 21 cases all but 3 had shown the clinical evidences of aortic leakage.There were 55 other cases of aortic endocarditis among the 930 autopsiesin which the etiologic factor was an antecedent acute endocarditis (historyof infections and involvement of other valves, 21 cases), and others in which thesclerosis was general, involved the whole aorta, and was ass

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1911

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